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SLC16A9 (基因名), Monocarboxylate transporter 9 (蛋白名), MOT9_HUMAN.
产品名称:

Human SLC16A9/ Monocarboxylate transporter 9 Recombinant Protein

货号:
-
商标:
EIAab®
监管等级:
别名:

Solute carrier family 16 member 9, MCT 9, C10orf36, MCT9

序列号:
Q7RTY1
来源:
E.coli
种属:
Human
标签:
His
纯度:
>90% by SDS-PAGE
浓度:
Reconstitution Dependent
形态:
Liquid
内毒素水平:
Please contact protein@eiaab.com The technician for more information.
应用:
存储缓冲液:
50mM NaH2PO4, 500mM NaCl Buffer with 500mM Imidazole, 10%glycerol(PH8.0)
存储:
Store at -20°C. (Avoid repeated freezing and thawing.)
研究领域:
-
Human SLC16A9 Protein
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Human SLC16A9 Protein
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产品说明书
说明书: 下载说明书
MSDS: MSDS


基因位点
Cytogenetic band: 10q21.2 by HGNC 10q21.2 by Entrez Gene 10q21.2 by Ensembl
SLC16A9 Gene in genomic location: bands according to Ensembl, locations according to GeneLoc (and/or Entrez Gene and/or Ensembl if different)
基因位点
通用注释


亚单元:
N/A


功能:
Proton-linked monocarboxylate transporter. May catalyze the transport of monocarboxylates across the plasma membrane.


亚细胞位置:
Cell membrane Multi-pass membrane protein


该产品尚未在任何出版物中被引用。

[1].
"Human metabolic individuality in biomedical and pharmaceutical research."

[2].
"Replication of the five novel loci for uric acid concentrations and potential mediating mechanisms."

[3].
"A genome-wide perspective of genetic variation in human metabolism."

[4].
"Meta-analysis of 28,141 individuals identifies common variants within five new loci that influence uric acid concentrations."

[5].
"Common polymorphisms influencing serum uric acid levels contribute to susceptibility to gout, but not to coronary artery disease."

[6].
"A scan of chromosome 10 identifies a novel locus showing strong association with late-onset Alzheimer disease."

[7].
"The SLC16 gene family-from monocarboxylate transporters (MCTs) to aromatic amino acid transporters and beyond."

[8].
"Complete sequencing and characterization of 21,243 full-length human cDNAs."

[9].
"Genome-wide CRISPR screen identifies HNRNPL as a prostate cancer dependency regulating RNA splicing."

[10].
"An atlas of genetic influences on human blood metabolites."
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