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首页  >  重组蛋白  >  Human ACO2 Recombinant Protein
ACO2 (基因名), Aconitate hydratase, mitochondrial (蛋白名), ACON_HUMAN.
产品名称:

Human ACO2/ Aconitate hydratase, mitochondrial Recombinant Protein
线粒体乌头酸水合

货号:

R1263h

商标:
EIAab®
监管等级:
别名:

Citrate hydro-lyase, Aconitase

序列号:
Q99798
来源:
E.coli
种属:
Human
标签:
His
纯度:
>90% by SDS-PAGE
浓度:
Reconstitution Dependent
形态:
Liquid
内毒素水平:
Please contact protein@eiaab.com The technician for more information.
应用:
存储缓冲液:
50mM NaH2PO4, 500mM NaCl Buffer with 500mM Imidazole, 10%glycerol(PH8.0)
存储:
Store at -20°C. (Avoid repeated freezing and thawing.)
研究领域:
-
Human ACO2 Protein
规格 & 价格: cart
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Human ACO2 Protein
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产品说明书
说明书: 下载说明书
MSDS: MSDS


R&D 技术数据
更多信息,请参阅手册,或联系我们的技术支持: tech@eiaab.com.
基因位点
Cytogenetic band: 22q13.2 by HGNC 22q13.2 by Entrez Gene 22q13.2 by Ensembl
ACO2 Gene in genomic location: bands according to Ensembl, locations according to GeneLoc (and/or Entrez Gene and/or Ensembl if different)
基因位点
通用注释


亚单元:
Monomer.


功能:
Catalyzes the isomerization of citrate to isocitrate via cis-aconitate.


亚细胞位置:
Mitochondrion


该产品尚未在任何出版物中被引用。

[1].
"The DNA sequence of human chromosome 22."

[2].
"Characterization of the human mitochondrial aconitase gene (ACO2)."

[3].
"Mutations in the tricarboxylic acid cycle enzyme, aconitase 2, cause either isolated or syndromic optic neuropathy with encephalopathy and cerebellar atrophy."

[4].
"Infantile cerebellar-retinal degeneration associated with a mutation in mitochondrial aconitase, ACO2."

[5].
"Genetic variants in nuclear-encoded mitochondrial genes influence AIDS progression."

[6].
"A genome annotation-driven approach to cloning the human ORFeome."

[7].
"The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)."

[8].
"Purification and partial amino acid sequence of human aconitase."

[9].
"RNA-binding activity of TRIM25 is mediated by its PRY/SPRY domain and is required for ubiquitination."

[10].
"A Map of Human Mitochondrial Protein Interactions Linked to Neurodegeneration Reveals New Mechanisms of Redox Homeostasis and NF-κB Signaling."
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