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首页  >  重组蛋白  >  Human ACAD9 Recombinant Protein
ACAD9 (基因名), Acyl-CoA dehydrogenase family member 9, mitochondrial (蛋白名), ACAD9_HUMAN.
产品名称:

Human ACAD9/ Acyl-CoA dehydrogenase family member 9, mitochondrial Recombinant Protein

货号:
-
商标:
EIAab®
监管等级:
别名:

ACAD-9

序列号:
Q9H845
来源:
E.coli
种属:
Human
标签:
His
纯度:
>90% by SDS-PAGE
浓度:
Reconstitution Dependent
形态:
Liquid
内毒素水平:
Please contact protein@eiaab.com The technician for more information.
应用:
存储缓冲液:
50mM NaH2PO4, 500mM NaCl Buffer with 500mM Imidazole, 10%glycerol(PH8.0)
存储:
Store at -20°C. (Avoid repeated freezing and thawing.)
研究领域:
Cardiovascular
Human ACAD9 Protein
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Human ACAD9 Protein
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产品说明书
说明书: 下载说明书
MSDS: MSDS


基因位点
Cytogenetic band: 3q21.3 by HGNC 3q21.3 by Entrez Gene 3q21.3 by Ensembl
ACAD9 Gene in genomic location: bands according to Ensembl, locations according to GeneLoc (and/or Entrez Gene and/or Ensembl if different)
基因位点
通用注释


亚单元:
Part of the mitochondrial complex I assembly (MCIA) complex. The complex comprises at least TMEM126B, NDUFAF1, ECSIT, and ACAD9 (By similarity). Interacts with NDUFAF1 and ECSIT (PubMed:20816094).


功能:
Required for mitochondrial complex I assembly (PubMed:20816094, PubMed:24158852). Has a dehydrogenase activity on palmitoyl-CoA (C16:0) and stearoyl-CoA (C18:0). It is three times more active on palmitoyl-CoA than on stearoyl-CoA. However, it does not play a primary role in long-chain fatty acid oxidation in vivo (PubMed:20816094, PubMed:24158852). Has little activity on octanoyl-CoA (C8:0), butyryl-CoA (C4:0) or isovaleryl-CoA (5:0).


亚细胞位置:
Mitochondrion


该产品尚未在任何出版物中被引用。

[1].
"Cloning and functional characterization of ACAD-9, a novel member of human acyl-CoA dehydrogenase family."

[2].
"Acyl-CoA dehydrogenase 9 is required for the biogenesis of oxidative phosphorylation complex I."

[3].
"Exome sequencing identifies ACAD9 mutations as a cause of complex I deficiency."

[4].
"A new genetic disorder in mitochondrial fatty acid beta-oxidation: ACAD9 deficiency."

[5].
"ACAD9, a complex I assembly factor with a moonlighting function in fatty acid oxidation deficiencies."

[6].
"Riboflavin-responsive oxidative phosphorylation complex I deficiency caused by defective ACAD9: new function for an old gene."

[7].
"Genetic variants in nuclear-encoded mitochondrial genes influence AIDS progression."

[8].
"Carnitine palmitoyltransferase I and Acyl-CoA dehydrogenase 9 in retina: insights of retinopathy in mitochondrial trifunctional protein defects."

[9].
"Acyl-CoA dehydrogenase 9 (ACAD 9) is the long-chain acyl-CoA dehydrogenase in human embryonic and fetal brain."

[10].
"Human acyl-CoA dehydrogenase-9 plays a novel role in the mitochondrial beta-oxidation of unsaturated fatty acids."
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