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YWHAE (基因名), 14-3-3 protein epsilon (蛋白名), 1433E_HUMAN.
产品名称:

Human YWHAE/ 14-3-3 protein epsilon Recombinant Protein
14-3-3蛋白ε

货号:

R14290h

商标:
EIAab®
监管等级:
别名:

14-3-3E

序列号:
P62258
来源:
E.coli
种属:
Human
标签:
His
纯度:
>90% by SDS-PAGE
浓度:
Reconstitution Dependent
形态:
Liquid
内毒素水平:
Please contact protein@eiaab.com The technician for more information.
应用:
存储缓冲液:
50mM NaH2PO4, 500mM NaCl Buffer with 500mM Imidazole, 10%glycerol(PH8.0)
存储:
Store at -20°C. (Avoid repeated freezing and thawing.)
研究领域:
Cancer
Human YWHAE Protein
规格 & 价格: cart
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Human YWHAE Protein
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产品说明书
说明书: 下载说明书
MSDS: MSDS
在线询价


R&D 技术数据
更多信息,请参阅手册,或联系我们的技术支持: tech@eiaab.com.
基因位点
Cytogenetic band: 17p13.3 by HGNC 17p13.3 by Entrez Gene 17p13.3 by Ensembl
YWHAE Gene in genomic location: bands according to Ensembl, locations according to GeneLoc (and/or Entrez Gene and/or Ensembl if different)
基因位点
通用注释


亚单元:
(Microbial infection) Interacts with HCV core protein.


功能:
Adapter protein implicated in the regulation of a large spectrum of both general and specialized signaling pathways. Binds to a large number of partners, usually by recognition of a phosphoserine or phosphothreonine motif. Binding generally results in the modulation of the activity of the binding partner (By similarity). Positively regulates phosphorylated protein HSF1 nuclear export to the cytoplasm (PubMed:12917326).


亚细胞位置:
Nucleus Cytoplasm Melanosome Identified by mass spectrometry in melanosome fractions from stage I to stage IV.


该产品尚未在任何出版物中被引用。

[1].
"KLHL22 activates amino-acid-dependent mTORC1 signalling to promote tumorigenesis and ageing."

[2].
"Front-signal-dependent accumulation of the RHOA inhibitor FAM65B at leading edges polarizes neutrophils."

[3].
"Suppression of death-associated protein kinase 2 by interaction with 14-3-3 proteins."

[4].
"ACBD3 interaction with TBC1 domain 22 protein is differentially affected by enteroviral and kobuviral 3A protein binding."

[5].
"Centrosome-related genes, genetic variation, and risk of breast cancer."

[6].
"Deletion of YWHAE in a patient with periventricular heterotopias and pronounced corpus callosum hypoplasia."

[7].
"Functional identification of a novel 14-3-3 epsilon splicing variant suggests dimerization is not necessary for 14-3-3 epsilon to inhibit UV-induced apoptosis."

[8].
"Further molecular and clinical delineation of co-locating 17p13.3 microdeletions and microduplications that show distinctive phenotypes."

[9].
"Lysine acetylation targets protein complexes and co-regulates major cellular functions."

[10].
"SLITRK1 binds 14-3-3 and regulates neurite outgrowth in a phosphorylation-dependent manner."
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