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MFHAS1 (GeneName), Malignant fibrous histiocytoma-amplified sequence 1 (ProteinName), MFHA1_HUMAN.
Product Name:

Human MFHAS1/ Malignant fibrous histiocytoma-amplified sequence 1 ELISA Kit

Cat.#:
-
Brand:
EIAab®
Regulatory Status:
Alternative:

Malignant fibrous histiocytoma-amplified sequence with leucine-rich tandem repeats 1, MASL1

Detection Method:
ELISA
Specificity:
Natural and recombinant human Malignant fibrous histiocytoma-amplified sequence 1
Sample Type:
Serum, plasma, tissue homogenates, cell culture supernates and other biological fluids
Sample Data:
Research Area:
-
Human MFHAS1 ELISA Kit
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Human MFHAS1 ELISA Kit
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Product Datasheets


General Annotation


Sub Unit:
N/A


Function:
N/A


Subcellular Location:
N/A


This product has not yet been referenced specifically in any publications.

[1].
"Identification of a novel gene, MASL1, within an amplicon at 8p23.1 detected in malignant fibrous histiocytomas by comparative genomic hybridization."

[2].
"GTP binding controls complex formation by the human ROCO protein MASL1."

[3].
"High Glucose Stimulates Expression of MFHAS1 to Mitigate Inflammation via Akt/HO-1 Pathway in Human Umbilical Vein Endothelial Cells."

[4].
"Ubiquitylation of MFHAS1 by the ubiquitin ligase praja2 promotes M1 macrophage polarization by activating JNK and p38 pathways."

[5].
"MFHAS1 suppresses TLR4 signaling pathway via induction of PP2A C subunit cytoplasm translocation and inhibition of c-Jun dephosphorylation at Thr239."

[6].
"MFHAS1 Is Associated with Sepsis and Stimulates TLR2/NF-κB Signaling Pathway Following Negative Regulation."

[7].
"MASL1 induces erythroid differentiation in human erythropoietin-dependent CD34+ cells through the Raf/MEK/ERK pathway."

[8].
"Genome-wide association study in multiple human prion diseases suggests genetic risk factors additional to PRNP."

[9].
"Human leucine-rich repeat proteins: a genome-wide bioinformatic categorization and functional analysis in innate immunity."

[10].
"Personalized smoking cessation: interactions between nicotine dose, dependence and quit-success genotype score."
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