ACRBP (GeneName), Acrosin-binding protein (ProteinName), ACRBP_PIG.
Product Name:
Pig ACRBP/ Acrosin-binding protein ELISA Kit
Cat.#:
-
Brand:
EIAab®
Regulatory Status:
Alternative:
Proacrosin-binding protein sp32
Detection Method:
ELISA
Specificity:
Natural and recombinant pig Acrosin-binding protein
Sample Type:
Serum, plasma, tissue homogenates, cell culture supernates and other biological fluids
Sample Data:
Research Area:
-
- Data
- Citations
- Publication
- Sequence / 3D
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General Annotation
Sub Unit:
Binds specifically to the 55- and 53-kDa proacrosins and the 49-kDa acrosin intermediate, but is not capable of binding 43-kDa acrosin intermediate and 35-kDa mature acrosin.
Function:
May be involved in packaging and condensation of the acrosin zymogen in the acrosomal matrix via its association with proacrosin.
Subcellular Location:
Secreted
Cytoplasmic vesicle
Secretory vesicle
Acrosome
Valid Sequence:
This product has not yet been referenced specifically in any publications.
[1].
"Identification of proacrosin binding protein sp32 precursor as a human cancer/testis antigen."
[2].
"Identification of an HLA-A24-restricted OY-TES-1 epitope recognized by cytotoxic T-cells."
[3].
"The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)."
[4].
"OY-TES-1 may regulate the malignant behavior of liver cancer via NANOG, CD9, CCND2 and CDCA3: a bioinformatic analysis combine with RNAi and oligonucleotide microarray."
[5].
"Cancer testis antigen OY-TES-1: analysis of protein expression in ovarian cancer with tissue microarrays."
[6].
"Down-regulation of cancer/testis antigen OY-TES-1 attenuates malignant behaviors of hepatocellular carcinoma cells in vitro."
[7].
"A human interactome in three quantitative dimensions organized by stoichiometries and abundances."
[8].
"Cancer testis antigen OY-TES-1 expression and serum immunogenicity in colorectal cancer: its relationship to clinicopathological parameters."
[9].
"Knockdown of OY-TES-1 by RNAi causes cell cycle arrest and migration decrease in bone marrow-derived mesenchymal stem cells."
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